My first post in 11 years.

[AlextheBitcoiner]

Wow. So many broke and jealous people here.

Sad!

[AlextheBitcoiner]

And what do you want to do after such a long time?

Hookers. Lots of hookers.

[AlextheBitcoiner]

Hello, I am back... what did I miss? 

After 11 years of HODLing, I am now writing to you from my new yacht, with a superstar model on my lap.

I do not have a high school degree.

Alex

Your intonation resemble that of a certain self-righteous professor who believe that everyone except him is illiterate. He is so boastful yet have nothing tangible to show for that high level of ego. I doubt there is anyone that have every responded to his trolling posts that have not receieve his abusive explosion and abuses. I have a strong feeling that you have something in common with that fellow.

If you have been holding Bitcoin for 11 years and possibly in your yatch, we would have known you or better still, drop some proofs to substantiate your bogus claim.

I literally dropped out. LOL. I'll make you a bet though, I'll send you proof. But if I don't have $1.8m in my BTC address, you need to send me $1K BTC. Deal or no deal?

[goldkingcoiner]

I do not have a high school degree.

This is the only part that's true.

And it is not the flex you think it is, even if you were really rich...

[BADecker]

@OP

You must have been away at sword school. You learned how to fence. Then you came back here and made a post (fence post).

 

[Vod]

if you have a supermodel on your lap but she isnt entertaining you, so you come to this forum.... its time to upgrade your model

lol.  Tell us you are impotent without telling us you are impotent. 

Seriously OP - congrats!  I remember your username and I actually tried the sailing thing too - did not agree with me. 

[breske]

there is a saying 1 pic = 1000 words

or it seems you just woke up and realised it was a great dream

[BADecker]

there is a saying 1 pic = 1000 words

or it seems you just woke up and realised it was a great dream

Well, will you remember any of this after you die?

A hundred years (if you live that long), and then it is all gone.


Neuroscientists just found a hidden protein switch in your brain that reverses aging and memory loss


https://www.linkedin.com/posts/lanson-burrows-jones-jr-49161651_neuroscientists-just-found-a-hidden-protein-activity-7366952717312823296-Psye/
Neuroscientists just found a hidden protein switch in your brain that reverses aging and memory loss. A new Nature Aging study just shattered the story of brain aging being inevitable. Researchers at UCSF discovered a single protein—FTL1 (ferritin light chain 1)—that acts like a on/off switch inside the hippocampus, the seat of learning and memory.

When scientists blocked FTL1 in aged mice, something extraordinary happened:
   •   Neurons sprouted new branches
   •   Synapses thickened
   •   Memories sharpened

The old brains didn’t just stop aging.
They became younger.

The reverse was also true: increase FTL1 in young mice, and their neurons collapsed into early aging. Dendrites shrank. Cognition declined. Their brains aged on fast-forward.

This is not about killing neurons.
It’s about the electrical and metabolic complexity that makes memory possible: dendritic architecture, iron regulation, and hippocampal energy.

And when the protein switch was flipped, decline wasn’t slowed.
It was reversed.

“It is truly a reversal of impairments … much more than merely delaying or preventing symptoms.” — Saul Villeda, UCSF

The data showed:
   •   FTL1 rises with age, correlating with memory loss
   •   Reducing FTL1 restored dendritic branching and synaptic signaling
   •   Overexpressing FTL1 triggered oxidative stress and synaptic collapse
   •   Boosting NADH metabolism rescued cognition, even in high-FTL1 states

This discovery reframes aging itself.
It isn’t just wear and tear.
It is programmable biology.
A hidden code.
A protein switch waiting to be turned.

The UCSF study used NADH in a targeted way inside neurons to rescue FTL1-induced decline. This Supplement has shown to support general brain energy metabolism, but they are not the same as directly switching off FTL1 like this protein in study but are avaliable today! NADH (nicotinamide adenine dinucleotide + hydrogen) is already available as a supplement. It’s essentially the reduced form of NAD⁺, the critical molecule your cells use for energy metabolism, mitochondrial function, and brain activity.

Imagine what this could mean for Alzheimer’s, dementia, Parkinson’s.
For every family who has watched a loved one fade away.
For decades we believed aging was inevitable.
That memory fades because neurons wear out.
That decline is irreversible.
If memory loss is not destiny but design—
then design can be rewritten.
...

[Johnlomape]

there is a saying 1 pic = 1000 words

or it seems you just woke up and realised it was a great dream

Well, will you remember any of this after you die?

A hundred years (if you live that long), and then it is all gone.


Neuroscientists just found a hidden protein switch in your brain that reverses aging and memory loss


https://www.linkedin.com/posts/lanson-burrows-jones-jr-49161651_neuroscientists-just-found-a-hidden-protein-activity-7366952717312823296-Psye/
Neuroscientists just found a hidden protein switch in your brain that reverses aging and memory loss. A new Nature Aging study just shattered the story of brain aging being inevitable. Researchers at UCSF discovered a single protein—FTL1 (ferritin light chain 1)—that acts like a on/off switch inside the hippocampus, the seat of learning and memory.

When scientists blocked FTL1 in aged mice, something extraordinary happened:
   •   Neurons sprouted new branches
   •   Synapses thickened
   •   Memories sharpened

The old brains didn’t just stop aging.
They became younger.

The reverse was also true: increase FTL1 in young mice, and their neurons collapsed into early aging. Dendrites shrank. Cognition declined. Their brains aged on fast-forward.

This is not about killing neurons.
It’s about the electrical and metabolic complexity that makes memory possible: dendritic architecture, iron regulation, and hippocampal energy.

And when the protein switch was flipped, decline wasn’t slowed.
It was reversed.

“It is truly a reversal of impairments … much more than merely delaying or preventing symptoms.” — Saul Villeda, UCSF

The data showed:
   •   FTL1 rises with age, correlating with memory loss
   •   Reducing FTL1 restored dendritic branching and synaptic signaling
   •   Overexpressing FTL1 triggered oxidative stress and synaptic collapse
   •   Boosting NADH metabolism rescued cognition, even in high-FTL1 states

This discovery reframes aging itself.
It isn’t just wear and tear.
It is programmable biology.
A hidden code.
A protein switch waiting to be turned.

The UCSF study used NADH in a targeted way inside neurons to rescue FTL1-induced decline. This Supplement has shown to support general brain energy metabolism, but they are not the same as directly switching off FTL1 like this protein in study but are avaliable today! NADH (nicotinamide adenine dinucleotide + hydrogen) is already available as a supplement. It’s essentially the reduced form of NAD⁺, the critical molecule your cells use for energy metabolism, mitochondrial function, and brain activity.

Imagine what this could mean for Alzheimer’s, dementia, Parkinson’s.
For every family who has watched a loved one fade away.
For decades we believed aging was inevitable.
That memory fades because neurons wear out.
That decline is irreversible.
If memory loss is not destiny but design—
then design can be rewritten.
...

If this works on mice, I would be expecting to see a positive result on human trial.
What a breakthrough for people suffering from memory loss and chronic abnormalities.

[BADecker]

there is a saying 1 pic = 1000 words

or it seems you just woke up and realised it was a great dream

Well, will you remember any of this after you die?

A hundred years (if you live that long), and then it is all gone.


Neuroscientists just found a hidden protein switch in your brain that reverses aging and memory loss


https://www.linkedin.com/posts/lanson-burrows-jones-jr-49161651_neuroscientists-just-found-a-hidden-protein-activity-7366952717312823296-Psye/
Neuroscientists just found a hidden protein switch in your brain that reverses aging and memory loss. A new Nature Aging study just shattered the story of brain aging being inevitable. Researchers at UCSF discovered a single protein—FTL1 (ferritin light chain 1)—that acts like a on/off switch inside the hippocampus, the seat of learning and memory.

When scientists blocked FTL1 in aged mice, something extraordinary happened:
   •   Neurons sprouted new branches
   •   Synapses thickened
   •   Memories sharpened

The old brains didn’t just stop aging.
They became younger.

The reverse was also true: increase FTL1 in young mice, and their neurons collapsed into early aging. Dendrites shrank. Cognition declined. Their brains aged on fast-forward.

This is not about killing neurons.
It’s about the electrical and metabolic complexity that makes memory possible: dendritic architecture, iron regulation, and hippocampal energy.

And when the protein switch was flipped, decline wasn’t slowed.
It was reversed.

“It is truly a reversal of impairments … much more than merely delaying or preventing symptoms.” — Saul Villeda, UCSF

The data showed:
   •   FTL1 rises with age, correlating with memory loss
   •   Reducing FTL1 restored dendritic branching and synaptic signaling
   •   Overexpressing FTL1 triggered oxidative stress and synaptic collapse
   •   Boosting NADH metabolism rescued cognition, even in high-FTL1 states

This discovery reframes aging itself.
It isn’t just wear and tear.
It is programmable biology.
A hidden code.
A protein switch waiting to be turned.

The UCSF study used NADH in a targeted way inside neurons to rescue FTL1-induced decline. This Supplement has shown to support general brain energy metabolism, but they are not the same as directly switching off FTL1 like this protein in study but are avaliable today! NADH (nicotinamide adenine dinucleotide + hydrogen) is already available as a supplement. It’s essentially the reduced form of NAD⁺, the critical molecule your cells use for energy metabolism, mitochondrial function, and brain activity.

Imagine what this could mean for Alzheimer’s, dementia, Parkinson’s.
For every family who has watched a loved one fade away.
For decades we believed aging was inevitable.
That memory fades because neurons wear out.
That decline is irreversible.
If memory loss is not destiny but design—
then design can be rewritten.
...

If this works on mice, I would be expecting to see a positive result on human trial.
What a breakthrough for people suffering from memory loss and chronic abnormalities.

But it wouldn't work on Navy Seals. Why not? Because they are men (and a few women), but not mice. Most other people are mice, however.

[Johnlomape]

there is a saying 1 pic = 1000 words

or it seems you just woke up and realised it was a great dream

Well, will you remember any of this after you die?

A hundred years (if you live that long), and then it is all gone.


Neuroscientists just found a hidden protein switch in your brain that reverses aging and memory loss


https://www.linkedin.com/posts/lanson-burrows-jones-jr-49161651_neuroscientists-just-found-a-hidden-protein-activity-7366952717312823296-Psye/
Neuroscientists just found a hidden protein switch in your brain that reverses aging and memory loss. A new Nature Aging study just shattered the story of brain aging being inevitable. Researchers at UCSF discovered a single protein—FTL1 (ferritin light chain 1)—that acts like a on/off switch inside the hippocampus, the seat of learning and memory.

When scientists blocked FTL1 in aged mice, something extraordinary happened:
   •   Neurons sprouted new branches
   •   Synapses thickened
   •   Memories sharpened

The old brains didn’t just stop aging.
They became younger.

The reverse was also true: increase FTL1 in young mice, and their neurons collapsed into early aging. Dendrites shrank. Cognition declined. Their brains aged on fast-forward.

This is not about killing neurons.
It’s about the electrical and metabolic complexity that makes memory possible: dendritic architecture, iron regulation, and hippocampal energy.

And when the protein switch was flipped, decline wasn’t slowed.
It was reversed.

“It is truly a reversal of impairments … much more than merely delaying or preventing symptoms.” — Saul Villeda, UCSF

The data showed:
   •   FTL1 rises with age, correlating with memory loss
   •   Reducing FTL1 restored dendritic branching and synaptic signaling
   •   Overexpressing FTL1 triggered oxidative stress and synaptic collapse
   •   Boosting NADH metabolism rescued cognition, even in high-FTL1 states

This discovery reframes aging itself.
It isn’t just wear and tear.
It is programmable biology.
A hidden code.
A protein switch waiting to be turned.

The UCSF study used NADH in a targeted way inside neurons to rescue FTL1-induced decline. This Supplement has shown to support general brain energy metabolism, but they are not the same as directly switching off FTL1 like this protein in study but are avaliable today! NADH (nicotinamide adenine dinucleotide + hydrogen) is already available as a supplement. It’s essentially the reduced form of NAD⁺, the critical molecule your cells use for energy metabolism, mitochondrial function, and brain activity.

Imagine what this could mean for Alzheimer’s, dementia, Parkinson’s.
For every family who has watched a loved one fade away.
For decades we believed aging was inevitable.
That memory fades because neurons wear out.
That decline is irreversible.
If memory loss is not destiny but design—
then design can be rewritten.
...

If this works on mice, I would be expecting to see a positive result on human trial.
What a breakthrough for people suffering from memory loss and chronic abnormalities.

But it wouldn't work on Navy Seals. Why not? Because they are men (and a few women), but not mice. Most other people are mice, however.

If Navy Seals are not mice, I guess you have not set a  free WiFi trap for the ones that looks like mice LoL!
I still don't get it why scientists always assume that if a trial could work on mice, there is possibility of working on humans...maybe we are twinny mice physiologically without a tail.

[BADecker]

~

But it wouldn't work on Navy Seals. Why not? Because they are men (and a few women), but not mice. Most other people are mice, however.

If Navy Seals are not mice, I guess you have not set a  free WiFi trap for the ones that looks like mice LoL!
I still don't get it why scientists always assume that if a trial could work on mice, there is possibility of working on humans...maybe we are twinny mice physiologically without a tail.

Science has found by trial and error that people and mice react to testing the same way.

[Jet Cash]

I've been a Bitcoiner and HODLer for almost 10 years, and I own a yacht. Unfortunately it's only a pond yacht. I do have a hot chick though, but she is 70.

[Johnlomape]

I've been a Bitcoiner and HODLer for almost 10 years, and I own a yacht. Unfortunately it's only a pond yacht. I do have a hot chick though, but she is 70.

Having a septuagenarian as a chick(lovely wife) is sweeter than altcoins. Being a Bitcoiner for 10 years shows your persistency experiencing different phases of the market trend.
For you to be a Bitcoiner that owned a yacht and flexes it on a pond, that's simply humility to teach others to hold their horses! Your words inspires like a compass guiding a lost boat.

[imstillthebest]

nah, you already made 49 post plus this year wasn't your recent but it was last year according in your post history. this thread still fits on me, because this is my first post after a long break. nice to be back in here  .