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Author Topic: Economic Totalitarianism  (Read 345644 times)
TPTB_need_war
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September 14, 2015, 12:16:16 AM
Last edit: September 14, 2015, 06:35:19 AM by TPTB_need_war
 #1221

Treating the myelin is myopic. Must treat the underlying cause which is gut dysbiosis. I am now very sure of this because I've correlated the pain in my small intestine and later again the traverse colon to the rise in the M.S. effects. And it correlates very well within hours for the small intestine and then again within 2 - 3 days of eating any grains or high Omega-6 meat. Also correlated to when I have loose stools instead of healthy stools.

http://gutcritters.com/part-four-dietary-fat-leaky-gut-endotoxemia-and-heart-disease/

Quote
Polyunsaturated: These types of fats have two or more double bonds and therefore lack four or more hydrogen atoms. Like monounsaturated fats, these double bonds are kinked so they don’t pack together well making them liquid even when refrigerated. Polyunsaturates can be further classified by the position of their first double bond in relation to their omega end. Polyunsaturated fats are called omega-6 fatty acids when their first double bond is in the sixth position from this position. Vegetable oils are very high in omega 6s.

Polyunsaturates with their first double bond from the third position of the omega end are called omega-3 fatty acids. Fish and flaxseed oils are both omega-3 oils yet differentiated by the length of carbon atoms they contain and their unsaturation.

These types of oils are extremely delicate and prone to oxidation when subjected to heat, light, pressure and free radicals in the body. They should never be used for high temperature cooking. Unfortunately, that is exactly what many vegetable oils are used for. They are routinely utilized in fast-food chains, restaurants and are common in processed foods.

All fats are combinations of different fatty acids. Canola oil, for example, is 62% monounsaturated fat, 6% saturated fat and 32% polyunsaturated fat. Butter fat is 56% saturated fat, 29% monounsaturated fat and 32% polyunsaturated fat.

Trans fats, implicated in both heart disease and cancer, are manufactured fats. They are made from polyunsaturated vegetable oils after the partial addition of hydrogen atoms to empty spots on their carbon chain. Olive oil and lard can also be subjected to partial hydrogenation to extend shelf life. Any lard you see that is not refrigerated is partially hydrogenated. Because hydrogenation straightens out the carbon chain, they have similar physical, although by no means biological, characteristics to natural animal fats. These are true Frankenfoods and should be avoided at all costs if you want to avoid a heart attack or stroke.

Your cells will reflect the type of fat you eat. Lipid peroxidation is the degradation of fats by oxidants leading to their damage and is not something you want happening to fats that are incorporated into your cellular structures. Of the fats mentioned, saturated fats are the least susceptible to this process.

Polyunsaturated oils, however, both omega 6 and omega 3, are particularly prone to lipid peroxidation by virtue of their missing hydrogen atoms. Omega 6 fatty acids are also inflammatory in excess.

While extremely delicate, omega 3 oils reduce inflammatory responses and are good for you as long as inflammatory stress in the liver is not an issue. Omega 3s subjected to oxidation can be very damaging.

How do we know this? Because the fastest way to cause alcohol-induced liver injury in an animal model is to feed them fish oil along with their Hooch. If you are prone to binge drinking, I really do not recommend that you wash down your fish or fish-oil capsules with loads of alcohol.

The history:

https://en.wikipedia.org/wiki/Cottonseed_oil#Economic_History

Quote
The by-product of cotton processing, cottonseed was considered virtually worthless before the late 19th century. While cotton production expanded throughout the 17th, 18th, and mid 19th centuries, a largely worthless stock of cottonseed grew. Although some of the seed was used for planting, fertilizer, and animal feed, the majority was left to rot or was illegally dumped into rivers.

In the 1820s and 1830s Europe experienced fats and oils shortages due to rapid population expansion during the Industrial Revolution and the English blockade during the Napoleonic Wars. The increased demand for fats and oils, coupled with a decreasing supply caused prices to rise sharply. Consequently, many Europeans could not afford to buy the fats and oils they had used for cooking and for lighting. Many United States entrepreneurs tried to take advantage of the increasing European demand for oils and America’s increasingly large supply of cottonseed by crushing the seed for oil. But separating the seed hull from the seed meat proved difficult and most of these ventures failed within a few years. This problem was resolved in 1857, when William Fee invented a huller, which effectively separated the tough hulls from the meats of cottonseed. With this new invention, cottonseed oil began to be used for illumination purposes in lamps to supplement increasingly expensive whale oil and lard. But by 1859, this use came to end as the petroleum industry emerged.

Cottonseed oil then began to be used illegally to fortify animal fats and lards. Initially, meat packers secretly added cottonseed oil to the pure fats, but this practice was uncovered in 1884. Armour and Company, an American meatpacking and food processing company, sought to corner the lard market and realized that it had purchased more lard than the existing hog population could have produced. A congressional investigation followed, and legislation was passed that required products fortified with cottonseed oil to be labeled as ‘‘lard compound.” Similarly, cottonseed oil was often blended with olive oil. Once the practice was exposed, many countries put import tariffs on American olive oil and Italy banned the product completely in 1883. Both of these regulatory schemes depressed cottonseed oil sales and exports, once again creating an oversupply of cottonseed oil, which decreased its value.

It was cottonseeds depressed value that lead a newly formed Procter & Gamble to utilize its oil. The Panic of 1837 caused the two brothers-in-law to merge their candlestick and soap manufacturing businesses in an effort to minimize costs and weather the bear market. Looking for a replacement for expensive animal fats in production, the brothers finally settled on cottonseed oil. Procter & Gamble cornered the cottonseed oil market to circumvent the meat packer's monopoly on the price. But as electricity emerged, the demand for candles decreased. Procter and Gamble then found an edible use for cottonseed oil. Through patented technology, the brothers were able to hydrogenate cottonseed oil and develop a substance that closely resembled lard. In 1911, Procter & Gamble launched an aggressive marketing campaign to publicize its new product, Crisco, a vegetable shortening that could be used in place of lard. Crisco placed ads in major newspapers advertising that the product was "easier on digestion...a healthier alternative to cooking with animal fats. . . and more economical than butter.” The company also gave away free cookbooks, with every recipe calling for Crisco. By the 1920s the company developed cookbooks for specific ethnicities in their native tongues. Additionally, Crisco starting airing radio cooking programs. Similarly, in 1899 David Wesson, a food chemist, developed deodorized cottonseed oil, Wesson cooking oil. Wesson Oil also was marketed heavily and became quite popular too.

Terry Whals protocol is wrong on eating seeds and slightly wrong to emphasize Omega-3 too much, rather should emphasize minimizing Omega-6 and not overdoing protein intake:

http://paleozonenutrition.com/2012/02/08/a-new-experiment-dr-wahls-super-nutrient-paleo-diet-9-cups-veggies-a-day/

Quote
Part of the nerve cell that is damaged in people with MS is the myelin sheath, a fatty electrically insulating layer along the axon. Myelin is about 70% fatty acids and Omega 3 is a major building block.

...

Omega 3 rich foods, ... wild fish and seafood. And you could add fish oil.

Here is why:

http://gutcritters.com/intestinal-alkaline-phosphatase-gut-health/

Quote
Intestinal alkaline phosphatase (IAP) is a brush-border enzyme expressed by absorptive cells (enterocytes) of the small intestine. It is secreted into both the lumen, or interior, of the digestive tract as well as the basolateral or systemic end.

Unlike gut flora, highest concentrations of IAP are found in the first section of the small intestine (duodenum) and decline the further down the digestive tract you go.

IAP secretion, just like any other small intestinal enzyme, is dependent on the health of enterocytes comprising what is known as the brush border. Intestinal cells that are chronically inflamed are by definition unhealthy, which is a major reason why those battling small intestinal dysbiosis are often deficient in these enzymes.

As you know, inflammation can be due to a number of factors: gluten, acetaldehyde, unsaturated fatty acids, enzyme inhibitors, gut infections, drugs, yeast overgrowth, thin to non-existent mucus layer, viruses, etc.. Whatever the cause, inflammatory cytokines will affect not only the shape of these cells and the tight junction proteins that bind them together, but their ability to properly secrete enzymes.

IAP has some very important functions. First, it’s involved in regulating secretion of bicarbonate in the duodenum.

Bicarbonate helps to neutralize the very acidic semi-digested food (chyme) entering the small intestine after passing through the stomach. Failure to raise pH here can lead to acidified chyme injuring cells lining this part of the digestive tract. That can increase inflammation and intestinal permeability.

But IAP’s most important role is detoxifying lipopolysaccharides (LPSs) derived from the cell wall components of gram-negative gut bacteria. (1) It is therefore an important defense against endotoxemia.

LPSs initiate inflammatory immune responses by binding to proteins known as toll-like receptors (TLRs), and in particular toll-like receptor 4 (TLR4). This in turn induces two separate inflammatory pathways.

The first is nuclear factor kappa B (NF-kB). NF-kB is a protein that regulates inflammatory immune responses, and chronic activation of this pathway has been linked to cancer and autoimmune disorders.

The second pathway initiated by TLR4 activation is release of tumor necrosis factor alpha (TNF-α). As you recall from this post, TNF-α is a very powerful and potentially destructive inflammatory cytokine.

The binding of TLR4 is a necessary condition for immune responses to gram-negative gut bacteria when they come into direct contact with intestinal epithelial cells and the submucosa. This inflammatory cascade is always accompanied by an increase in cortisol generation and synthesis via activation of the hypothalamic-pituitary-adrenal (HPA) axis, and by increasing intracellular expression of the enzyme 11β-hydroxysteroid dehydrogenase type 1 (11β-HSD1) via the cortisol-cortisone shunt.

Now this immune complex causes the release of IAP as a defensive mechanism. IAP’s ability to detoxify LPSs prevents further immune stimulation. IAP also prevents NF-kB from migrating to the nucleus of enterocytes. So by both detoxifying LPSs and halting NF-kB, IAP serves as a vital anti-inflammatory intestinal enzyme.

Conversely, cytokines like TNF-α and interleukin 1 beta (IL1-β) depress the secretion of IAP from enterocytes. Why this is so no one really knows. However, as mentioned above, oxidative stress no doubt has something to do with it. Very little is more stressful to gut cells than being subjected to the chronic onslaught of inflammatory cytokines.

Therefore as a general rule it’s safe to say that anything that chronically inhibits IAP intestinal expression will increase the risk of LPSs initiating inflammatory immune responses at the gut wall that can result in a leaky gut. In very severe cases sepsis can result should these bacterial components reach systemic circulation in appreciable amounts. At the very least, LPSs reaching the liver will provoke inflammation capable of damaging cells in that organ.

Dietary Fats and Intestinal Alkaline Phosphatase Expression

IAP expression is partly influenced by the type of fatty acids present in the small intestine after a meal. As I covered in this post, unlike short- and medium-chain fatty acids, long-chain fatty acids do not go directly to the liver absent a leaky gut:

“Longer-chain fatty acids…re-form into triacylglycerols within the enterocyte. Together with phospholipids, cholesterol and proteins, they form large particles within the absorptive cell. If there are lipopolysaccharides in the vicinity, these too hitch a ride on the newly formed lipoprotein vehicle or chylomicron.

As I wrote then, chylomicrons and all members of the cholesterol family bind to and inactivate LPSs with varying degrees of efficiency, which is a good thing as Homo Sapiens have been eating long-chain fatty acids for approximately 195,000 years, with most of these lipids coming from animal sources.

However, cholesterol isn’t the only biological substance that defends us against endotoxins. In animal models, IAP secretion has been consistently shown to rise in the presence of saturated fatty acids.

If as a normal consequence of absorbing these fats, LPSs come into direct contact with brush border cells, it’s not at all surprising that this enzyme would be excreted as part of an innate immune defense against these potential troublemakers.

But while saturated fatty acids increase the secretion of this anti-inflammatory enzyme, polyunsaturated fatty acids (PUFAs)–both from omega 6 and omega 3 sources–have been shown in both pigs and rodents to either not provoke IAP release, or actively suppress it.

This is a decidedly undesirable state of affairs.

LPSs carried across the gut wall by long-chain PUFAs are also inactivated once incorporated into chylomicrons. However, the failure to increase IAP secretion when LPSs either reach enterocytes, or slip between them when tight-junction proteins are compromised, inevitably causes immune activation. Couple this with the propensity of these fatty acids to easily oxidize and generate lipid peroxidation byproducts, and the risk of inducing leaky gut and endotoxemia goes through the proverbial roof.

Is it any wonder then that rodents chronically fed alcohol while simultaneously fed PUFAs experienced the liver damage I showed you in this post, while saturated fats were protective under the same chronic alcohol feeding?

In pigs fed either corn oil or beef tallow for four weeks, corn oil feeding consistently decreased a number of brush-border intestinal enzymes (4)

Foods high in Omega 6 to avoid like the plague:

http://www.healthaliciousness.com/articles/high-omega-6-foods.php

http://authoritynutrition.com/optimize-omega-6-omega-3-ratio/

http://www.nutritionj.com/content/9/1/10

Quote
Omega-3 (n-3) and omega-6 (n-6) fatty acids are two separate distinct families...Table 2 shows significant differences in n-6:n-3 ratios between grass-fed and grain-fed beef, with and overall average of 1.53 and 7.65 for grass-fed and grain-fed, respectively

http://gutcritters.com/part-four-dietary-fat-leaky-gut-endotoxemia-and-heart-disease/

Quote
In this post, I discussed how unlikely it is that normal digestion of long-chain fatty acids–whether saturated, monounsaturated or polyunsaturated—is the source of the pathogens initiating arterial plaque formation. If anything, I showed how protective chylomicrons are in preventing just that. This then leaves us with increased intestinal permeability as the most likely source of translocating pathogens.

While today’s post will focus on the role of dietary fat in “leaky gut”, I once again need to emphasize that once you have intestinal dysbiosis, ANY food you eat will increase the translocation of bacteria, yeast, larger food molecules, etc. into the bloodstream leading to the liver and to a smaller extent, systemic circulation. The more often you eat, the more frequently this happens.

Since small-gut dysbiosis increases hunger and cravings by its negative effects on gut hormone secretion and nutrient absorption, reigning in overeating can be challenging until dysbiosis is tamed via dietary change and resolution of bacterial and yeast overgrowth.

Increased intestinal permeability will allow long-chain fatty acids, that would normally only be incorporated into chylomicrons, access to the blood flowing to the liver also carrying with them antigens and bacteria that will provoke an immune response. As more fat is now reaching the liver, more cholesterol will be synthesized to export it. As I said, changes in cholesterol levels are a marker of endotoxemia, not the cause of it. I’ll have more to say about this in an upcoming post.

OK, let’s look more closely at the anatomy of the small intestinal gut wall:

...

We know that gluten opens these junctions. (2) Plant lectins are also disruptive. (3) Alcohol, especially binge drinking, also compromises intestinal integrity. (4) Small intestinal dysbiosis, and the inflammation that results from such an overgrowth, directly impacts these protein structures for the worse.

All oxidation from gut inflammation has the potential to affect these proteins. Oxidation is a normal process of cells that use oxygen to produce energy from various substrates, including those cells lining the intestinal tract. This process is called oxidative phosphorylation, and it would take an entire book to cover. Should you be interested, this Wikipedia entry is a good place to start. As this article makes clear, the gram-negative pathogen, E. coli, is quite adept at growing in an oxidative environment.

Most oxidation within cells will be harmlessly converted to water but not all. Two very harmful intermediate substances normally produced are superoxide anion and peroxide known collectively as reactive oxygen species or ROS. These are highly unstable agents. They have the potential to damage DNA, proteins, fats and intestinal cells, including those producing protective mucus. There are a number of built-in defenses that cells use to guard themselves against these harmful substances but suffice it to say that these defenses can be overwhelmed in times of intense free radical production as part of an immune response.

Anything that increases oxidation in the intestinal tract will also disrupt beneficial bacterial populations. Especially vulnerable are Lactobacillus species that predominate in the small intestine. These bacteria do not handle oxidation well, certainly not as well as gram-negative pathogens like E. coli. Bifidobacteria species in the colon are also negatively affected.

One substance that can be extremely oxidizing is fructose. Fructose forms half of the sugar molecule and can comprise anywhere from 42% to 90% of high-fructose corn syrup. We are well adapted to handling moderate amounts of it in its natural form where it comes packaged with fiber, antioxidants, vitamins and phytochemicals. Strip it of these protective substances during refining and we become far more prone to its ill effects. In large quantities, fructose produces lots of free radicals in those intestinal cells that are able to metabolize it because of its ability to rapidly degrade ATP to uric acid. (5)

Fructose, gluten, lectins and alcohol are not the only dietary components that increase oxidative stress in intestinal cells. Some fats do too.

You’ve all heard of saturated, monounsaturated, and polyunsaturated fats. The difference between these fats comes down to whether the carbon atoms that compose them contain double bonds.

Saturated: These fats do not have any double bonds between carbon atoms, and are saturated with hydrogen atoms. Saturated fats are very stable and not prone to oxidation when subjected to heat or free radicals in the body. For this reason, they are ideal for high-temperature cooking. Because they are straight in form, saturated-fatty-acid chains pack together readily and are solid or semisolid at room temperature. Saturated fats are found in animal fats and in tropical oils like palm and coconut oil.

Monounsaturated: These fats have one double bond which means two carbon atoms in the chain are double-bonded to each other. For this reason, they lack two hydrogen atoms. At the double-bond, these fats form a kink, so they don’t pack as easily as saturated fats. They are therefore liquid at room temperature although will congeal somewhat when refrigerated. While stable, these oils are more prone to oxidation than saturated fats. Oleic acid is a common form of monounsaturated fat found in our food and is the main component of olive oil. Almond, pecan, cashews, peanuts and avocados are also rich in oleic acid as is lard. Lard is 44% oleic acid, 42% saturated fat and 10% polyunsaturated fat. As fats are typically classified by the predominant fatty acid contained in them, lard should be classified as a monounsaturated fat, not a saturated one.

Polyunsaturated: These types of fats have two or more double bonds and therefore lack four or more hydrogen atoms. Like monounsaturated fats, these double bonds are kinked so they don’t pack together well making them liquid even when refrigerated. Polyunsaturates can be further classified by the position of their first double bond in relation to their omega end. Polyunsaturated fats are called omega-6 fatty acids when their first double bond is in the sixth position from this position. Vegetable oils are very high in omega 6s.

Polyunsaturates with their first double bond from the third position of the omega end are called omega-3 fatty acids. Fish and flaxseed oils are both omega-3 oils yet differentiated by the length of carbon atoms they contain and their unsaturation.

These types of oils are extremely delicate and prone to oxidation when subjected to heat, light, pressure and free radicals in the body. They should never be used for high temperature cooking. Unfortunately, that is exactly what many vegetable oils are used for. They are routinely utilized in fast-food chains, restaurants and are common in processed foods.

...

Any fat in the presence of gut wall dysfunction, along with protein and carbohydrate, will cause translocation of gut pathogens. The majority of these pathogens will end up in the liver creating oxidative damage and disease. A small portion will bypass the liver entirely and directly enter the bloodstream. In more advanced cases of “leaky gut” and subsequent hepatic damage, these pathogens will also escape the liver and enter systemic circulation. Cholesterol will try to neutralize these substances and repair the damage they cause in arteries, but along with other responding immune cells, form atheromas and fibrous caps. If unstable, these complexes can rupture producing a heart attack or stroke.

In populations where gut dysbiosis and endotoxemia are rampant, encouraging people to substitute highly reactive and inflammatory omega 6 polyunsaturated fats for saturated fat is nothing short of dietary madness and a denial of the basics of fatty acid structure and biochemistry.

The cause of heart disease is metabolic endotoxemia. Binge drinking, excess consumption of sugar, trans fats, overeating, omega 6 oils, tooth decay, respiratory infections, gluten, stress, aging, poor anti-oxidant status, cigarette smoking, etc. are all risk factors for cardiovascular disease because they can all negatively impact gut wall integrity and beneficial bacterial populations. Correcting dysbiosis through changes in diet and resolving bacterial and yeast infections while replenishing and maintaining beneficial gut flora populations is the only hope you have for preventing this potentially deadly disease.


http://gutcritters.com/small-intestinal-bacterial-overgrowth-part-seven-the-role-of-plant-lectins/

Quote
there are two families of lectins that have toxic effects in humans if they appear in our food.

Lectins found in legumes (beans and peanuts) and gluten grains are the most problematical. Whole-gluten grains like whole-wheat is a particularly rich source of a lectin called wheat-germ agglutinin or WGA for short. WGA is found in highest concentration in the germ of the plant. This makes the most evolutionary sense as the seed of the plant contains the genetic material necessary to propagate the species.

Lectins have shown very active biological properties when studied in animals and humans and entire books have been written on the subject. It is not my intention to summarize all these findings here. Rather, I want to concentrate on those factors that are directly relevant to acquiring small intestinal bacterial overgrowth.

But first, let me clear something up. Recently, there has been talk in the “Paleo Diet” and health blogosphere that concerns about lectin toxicity in the diet are overblown and should not unduly worry anyone because cooking inactivates them.

Yes, wet-heat cooking does indeed inactivate these pesticides. Subjecting beans to prolonged, high-heat cooking renders them a harmless and nutritious, albeit fiber-rich and gassy, source of food. And subjecting wheat-based foods to wet heat as in boiling pasta or noodles also inactivates WGA. So far so good.

However, dry-heat cooking methods as exists in baking or breakfast cereal manufacturing does not seem to inactivate them.

...

I need to make one other clarification before proceeding. The findings I refer to were conducted in animals using raw bean or wheat lectin which are pretty much interchangeable as they attach to the same gut structures and intestinal flora.

This obviously doesn’t mimic how humans consume these foods in real life. No one is eating uncooked legumes (except for peanuts) or gnawing on a stalk of wheat. If they are they have bigger problems to contend with.

But that doesn’t mean that these research findings are irrelevant. Far from it. While human exposure to these natural pesticides is at far lower concentrations than what was used in animal studies (or at least I hope so), the impact on gut health from low-grade, chronic exposure would be expected to be the same although not manifest as quickly.

In a culture where wheat is the “king” of grains and consumption of processed-wheat products is pervasive, exposure to lectins begins at a very early age and extends for a lifetime in most. Meal after meal, day after day, year after year and the damage begins to pile up.

Lectins, inflammatory gluten peptides, gluten opioids, adenosine and increased zonulin production really make this noxious grain and its siblings a true biohazard of the first order for the gut. Throw in other gut-destroying practices like repeated courses of antibiotics, drugs (both licit and illicit) and alcohol and the negative impact skyrockets.

Lectin’s known effects on gastrointestinal function

Lectins are extremely efficient at attaching themselves to the mucosa of the small intestine. The interior lining of our small intestine is called the sugar coat for a reason. Here, cells and bacteria present a surface coating that consists of sugar molecules attached to either protein (glycoprotein) or fat (glycolipid) creating a kind of “slime” that serves both a protective and attachment function.

In lectin-fed rodents, lectins bind to and strip away the mucous coat exposing the underlying cells to the contents of the lumen. They also inhibit repair by blocking mucus secreting cells from producing this important protective lubricant.

Rodents fed lectins are incapable of properly utilizing the protein content of their diets. If sufficient quantities of lectins are present in their food, the rate of protein breakdown and loss from their body exceeds the amount of protein taken in.

Lectins bind to the epithelial cells of the small intestine resulting in disfigurement and damage to the digestive brush border.

Lectins cause rapid cell division, growth and turnover in the cells lining the small intestine. Because of this, these cells are too immature to properly digest food or maintain proper intestinal barrier function.

In rodents, lectins consistently increase the size of the pancreas.

Lectins bind to and interfere with cells responsible for the production of various gut-satiety hormones. Satiety hormones tell your brain, gee, I’m really full and can’t eat another bite. When they aren’t produced or produced in lower amounts than normal, hunger is a constant companion. I’ll explore this topic in a future series on gut health and weight regulation.

Lectins bind to and interfere with the gut-associated lymphoid tissue or GALT system. This is the gut’s immune system that protects against infections from pathogens.

Finally, lectins have been shown to directly disturb gut flora populations in animals.

I want to put forward the hypothesis that lectins are uniquely toxic to our beneficial gut flora. As disturbed gut flora seems to be a necessary condition for the initiation of infection in the small intestine, this is not a minor issue.

This theory revolves around the fact that lectins, like WGA, have a high affinity for gram-positive bacteria. Beneficial Lactobacillus and Bifodobacterium species are of this type.

...

Bean lectins like phytohaemagglutinin or PHA which is found in red kidney beans and wheat germ agglutinin found in gluten grains are specifically designed to attach to cellular structures that express N-acetyglucosamine. If this name sounds familiar, it’s because the word glucosamine is part of it. Glucosamine is what a lot of you take to treat your aching joints.

Because peptidoglycan has two sugar components with one of them being N-acetyglucosamine, these lectins attach to it rather readily. Because the peptidoglycan area of gram-positive bacteria is large and exposed, it can be said that these types of lectins have a high affinity for them.

Conversely, because the peptidoglycan membrane in gram-negative bacteria is thinner and protected behind an outer lipopolysaccharide layer, these lectins do not bind to them or disturb them in any way.

...

The bad news is that these lectins are also harmful to beneficial gut flora.

Because gram-negative bacteria are not affected by these lectins, they are more likely to proliferate when beneficial gram-positive gut flora populations are reduced or absent.

In the book Plant Lectins we learn:

“Food and some of its components, particularly the lectins, may directly interact with the bacterial flora or, alternatively, they may indirectly affect bacterial proliferation in the small intestine through interference with the binding of selected species to epithelial tissues. Whichever of these two mechanisms operates, the end result is the potential inducement of selective proliferation of some species of bacteria in the digestive tract, including the small intestine.”

And in animals fed red kidney bean lectin:

“…concurrent with increased toxicity, there is a dramatic overgrowth of Escherichia coli in the small intestine of conventional rats fed on PHA-containing diets…Similar studies with other animal species have fully confirmed the existence of this causative relationship between the presence of PHA in the diet, E. coli overgrowth and toxicity. Although the mechanism of the selective overgrowth and how this affects nutritional efficiency is not clear, one possible mechanism is that the lectin-induced virulence of coliforms [gram-negative pathogens] in the small intestine of kidney bean-fed rats is the result of the elimination of competing species.”

I reiterate again that this is only a hypothesis and has yet to be proven true in humans because no one that I’m aware of has subjected this to clinical investigation.

If beans are a large part of your diet, make sure they are cooked at high temperatures for a sufficient length of time. Many people have been made ill eating undercooked red kidney beans. Don’t be one of them.

I don’t eat beans in restaurants or out of a can because I have no way of knowing how thoroughly they were cooked. The only beans I eat are those I cook myself, and I would recommend you do the same.

Avoid peanuts as they are high in lectins and dry roasting them does not inactivate these toxins.

As for wheat germ agglutinin, make sure your gluten-food product is subjected to wet-heat cooking. Ready-made processed gluten foods are best avoided. Hell, I think gluten grains are best avoided which is why I don’t eat them whether sprouted or fermented.

It is possible that sourdough fermentation may neutralize WGA but I haven’t read anything in the literature to that effect. If anyone out there has info on this, please share.

To counter the negative effects of lectins in the diet, consider supplementing with prebiotics and probiotics found in food or supplements.

Also consider taking a glucosamine pill with every meal containing beans or gluten grains. This will act as a decoy and bind lectins before they can do any damage to your mucosa, digestive cells, and friendly gut flora.


Edit: I think drastically increasing my cayenne pepper consumption is also a significant factor in being able to correct my gut dysbiosys:

http://www.globalhealingcenter.com/natural-health/benefits-of-cayenne-pepper/

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September 14, 2015, 01:10:19 AM
 #1222

I don't disagree with the suggestion to get the hell out of the house-of-cards banking system especially with near-zero interest rates, but the story about branch cash limits is sensationalism. Those have been around for 10-15 years at least, and my personal experience agrees with what the article said about getting as much cash as you want by calling ahead to order it or going to a bigger branch.

Too much crap on the web these days is basically clickbait in one form or another. That's what happens when you start being able to monetize eyeballs.

EDIT: I agree the push for negative interest rates threatens real limits, but that hasn't happened yet most places. The reports of such from Switzerland are mixed, and I'm not really sure who to believe, but I suspect some sensationalism there too.
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September 14, 2015, 06:07:17 AM
Last edit: September 14, 2015, 09:49:28 AM by TPTB_need_war
 #1223

smooth I agree. I do note that a recent international $6000 SWIFT transfer from some BTC I sold was held up by anti-money laundering policies. The sender had to call his bank and have them take some special actions to accelerate the transfer.

I was also impacted a several years ago when some wire transfers to a gold dealer in the USA were held for weeks and then sent back to the sender abroad. This caused a busted trade which impacted me to the tune of a $10,000 loss on the movement of the price interim.

I was once sent a 1052 troy.oz Comex bar by a dealer (tulving.com which is not defunct) in the USA with the serial # chiseled off and when I demanded restitution he refused to honor the movement in the price interim and took 1 month to replace the bar, thus causing me another huge loss.

I haven't accounted for how much money I lost from various antics of gold dealers, but suffice it to say it is in the $50,000-$100,000 range and it was more than I lost so far due to the government's AML crap. Which is another strong reason I want to make crypto-currency that I feel I can rely on, because I really fucking hate precious metals. I could see myself buying a few gold coins to fondle once in a while but not putting a significant portion of my networth (which is nearly 0 by now) in gold nor silver.

Thus my advice to not deal in gold & silver for a large % of your networth as I used to do. You will surely lose your networth as I did. That is a sure fire way to emulate me and end up bankrupt. I ended up bankrupt for numerous reasons (a lot of circumstance involving the devolution of my family life due to the choice to get involved with a filipina back in the mid-1990s that all came to fucked state by 2003 - 2008), such as being in such a bizarre state of psychosis after an ICU hospitalization for acute peptic ulcer in May 2012 which was the breaking point where my gut dysbiosis went bezerk, that I followed this promoter Graham Summers on the idea that China's stock market would imminently collapse and I tripled down on some options bets ended up losing $75,000 before I finally gave up. That was the reason I was in such a self-disciplinary mode that when rpietila told me several times from August 2012 to Jan 2013 that he was getting into Bitcoin and I gave him my thumbs up in January to sell silver and go all in, that I was unable to follow him in because I had made the decision that I didn't trust my mental state and with my failing health I would need to be ultra conservative. In hindsight, if I had invested in Bitcoin the $75,000 or so I had remaining in January 2013 then I'd have no financial problems now and probably would have been able to finance the medical attention that could have focused me sooner on how to cure M.S.. I didn't even realize I had M.S. until Spring 2015.

Even recently I thought I was following a Paleo diet by eating lots of meat and no grains. Turns out that all meat other than fish has very high Omega-6 (and no Omega-3 since it is not grass fed) and thus I was making my gut dysbiosis worse! That explains why I got worse over the past 3 months while my gf was cooking for me 3 meals a day. It reached the breaking point in August where I had to start fasting because my M.S. was progressing from remitting and relapsing to nearly continuous. I have realized the reason I coped reasonably well M.S. even though it was worsening, is because I was exercising so much. Terry Whals explains that exercise helps rebuild the myelin that is destroyed by M.S.. So I was constantly rebuilding and destroying, rebuilding and destroying. But that battle was wearing me down. I had reached a breaking point where I was going to cave in and start to lose motor function.

So any way back to the banking system. I agree the capital controls haven't really kicked in yet in terms of smaller amounts. And for larger amounts as long as you follow all the KYC procedures normally you are okay. Just never carry cash around because this can get seized and you can be declared a drug dealer, with the burden of proof on you to prove not. Note I've read from a reputable source (nestmann.com) that much of the circulating dollars have residue of cocaine on them.

But the powers that be have put all this FATCA and AML (Patriot Act) infrastructure in place for reason.

And OROBTC's point is appropriate. You have to get out while it is still possible to do so. By the end of 2017, it will be too late. Those structures will be activated by them because international capital will be on the move and the governments will be increasingly desperate.


Edit: in case I didn't make it clear enough. Gold and silver are useless without gold and silver dealers. Those dealers won't be reliable when you need them to be. The idea of trading coins in the underworld is a good way for you to lose an eyeball (as I did to some gang but for different reason) or some of your limbs. Gold and silver are not safe. It is dangerous shit like dynamite. Even keeping in your basement is dangerous. Buy gold and silver and enter the underworld. A prominent former US Treasury official quipped, "we will burn the fingertips of the gold bugs up to their armpits". I believe that might have been Robert Rubin.

Edit#2: I am a programmer, and do not have time to be a full-time investor. My mind is deep in other things. I can't keep up with things that others can do normally, such as little details about investments. This is why gold and silver are really problemmatic for me. For example, I have no place to store them where I live. Carrying them with me in my travels is entirely unrealistic. They have been nothing but a nightmare hassle and loss for me. I once had 18,000oz of silver. Now I have nothing.

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September 14, 2015, 07:03:57 AM
Last edit: September 14, 2015, 07:37:31 AM by TPTB_need_war
 #1224

All this health stuff is relevant to the upcoming Economic Totalitarianism because medical care systems are going to go bankrupt and fail. And because we are all making ourselves sick with the foods we eat. For example, did you pay attention to the red text in my prior post, with research confirming that grass fed meat has 5 times more Omega-3 than corn fed.

http://gutcritters.com/small-intestinal-bacterial-overgrowth-part-seven-the-role-of-plant-lectins/

Quote
...

But first, let me clear something up. Recently, there has been talk in the “Paleo Diet” and health blogosphere that concerns about lectin toxicity in the diet are overblown and should not unduly worry anyone because cooking inactivates them.

Yes, wet-heat cooking does indeed inactivate these pesticides. Subjecting beans to prolonged, high-heat cooking renders them a harmless and nutritious, albeit fiber-rich and gassy, source of food. And subjecting wheat-based foods to wet heat as in boiling pasta or noodles also inactivates WGA. So far so good.

However, dry-heat cooking methods as exists in baking or breakfast cereal manufacturing does not seem to inactivate them.

...

If beans are a large part of your diet, make sure they are cooked at high temperatures for a sufficient length of time. Many people have been made ill eating undercooked red kidney beans. Don’t be one of them.

I don’t eat beans in restaurants or out of a can because I have no way of knowing how thoroughly they were cooked. The only beans I eat are those I cook myself, and I would recommend you do the same.

Avoid peanuts as they are high in lectins and dry roasting them does not inactivate these toxins.

As for wheat germ agglutinin, make sure your gluten-food product is subjected to wet-heat cooking. Ready-made processed gluten foods are best avoided. Hell, I think gluten grains are best avoided which is why I don’t eat them whether sprouted or fermented.

It is possible that sourdough fermentation may neutralize WGA but I haven’t read anything in the literature to that effect. If anyone out there has info on this, please share.

To counter the negative effects of lectins in the diet, consider supplementing with prebiotics and probiotics found in food or supplements.

Also consider taking a glucosamine pill with every meal containing beans or gluten grains. This will act as a decoy and bind lectins before they can do any damage to your mucosa, digestive cells, and friendly gut flora.


Edit: I think drastically increasing my cayenne pepper consumption is also a significant factor in being able to correct my gut dysbiosys:

http://www.globalhealingcenter.com/natural-health/benefits-of-cayenne-pepper/

I was eating raw beans sometime in 2010 - 2012 before I got so ill. I was having some peripheral neuropathy effects before that due to others reasons (overusing antibiotics, probably still lingering high strain HPV infection, eating poorly and irradically, not getting sunlight being on the computer too much), but it wasn't until I did some really stupid things like this that caused my gut dysbiosys to go bezerk. And the acute peptic ulcer broke the camel's back because then I took 6000mg of antibiotics daily for 24 days (because the doctor messed up my first prescription and I couldn't handle the Clarythromycin as it made everything smell so horrible that I wanted to kill myself literally) and then I ended up with a severe fungal infection and I was munching on peanuts and cashews every day sitting on the computer and turns out those are so damn high in lectins! I did about everything possible wrong. It is very surprising that I am not dead.

Turns out that there is a way to eat bread that is nutritious and not harmful!

http://www.culturedfoodlife.com/can-sourdough-change-the-gluten-free-diet/

Quote
About ten years ago I went to a class on “How to Make Sourdough Bread.”  My daughter had gluten intolerance and we found that she could eat sprouted bread without the side effects created by regular bread. I had heard that sourdough bread achieved similar results to the sprouted bread, and I wanted to try it. What I learned shocked me. The man teaching the class explained that the process of making sourdough was an ancient art and one that had many benefits that we are unaware of today. Why do so many of us struggle with gluten today? There are all kinds of books and websites dedicated to gluten-free living, and rightfully so, because the bread we have today is very different from the bread we ate for hundreds of years. But why is gluten intolerance an epidemic in this day and age? What has changed?

Before the 1950’s, most bread bakeries ran two shifts of workers because the dough was fermented throughout the night with a long and slow process using a culture that contained the lactobacillus bacteria. This slow process was necessary for bread to be properly digested. In the process of making sourdough bread, the bran in the flour is broken down during the long rising time, releasing nutrients into the dough. Only when wheat gluten is properly fermented or sprouted (to learn more about sprouted breads click here) is it healthy for human consumption.  When not, it is potentially one of the most highly allergenic foods we eat. The phytic acid in grain needs to be 90% neutralized in order for the minerals to be absorbed by the human body. When you naturally ferment or sprout bread, you eliminate all phytic acid. About 90% of the phytic acid remains in breads made with instant yeasts, unless it is sprouted bread.

In their efforts to increase profits and speed up the the bread making process, bakers began using new techniques that took only three hours to make a loaf of bread – and now can even take only one hour. They used the new instant yeasts, which made the old way of making bread (using cultures and fermentation that not only help to preserve food, but also increase the nutrients available for our bodies) unnecessary.

During the making of sourdough bread, complex carbohydrates are broken down into more digestible simple sugars, and protein is broken down into amino acids. Enzymes develop during rising.  These enzymes are not lost while baking since the center of the loaf remains at a lower temperature than the crust. This fermentation, partly from lactobacillus, also allows for a bread that is lower on the glycemic index, thus making it better for those with blood sugar issues. The fermentation also helps restore the functioning of the digestive tract, resulting in proper assimilation and elimination.

These changes in our bread have had devastating effects on our gut. I believe that along with the overly processed foods, soil depletion, and the loss of fermentation and probiotic foods that heal and protect our bodies, our diets are wreaking havoc on our guts. This, in turn, is causing the rise in all kinds of food allergies. Our diets are a dim reflection of the nutrient-dense whole foods we used to eat years ago. Someone at a recent class asked why we are living longer if our diets are so bad. But this is actually not the case any more; we are not living longer, this trend has stopped. Not only that, the quality of our lives is in sad shape. How often do you see someone living vibrantly and without sickness or ailments?  It is increasingly becoming the exception and not the norm. Pharmaceuticals are the norm and not the exception, and food allergies and gut issues are rampant along with a host of other health issues. The average consumer is unaware of these changes in our food supply and then labels gluten and breads as the enemy, when they don’t realize the culprit is the dramatic changes in the actual process of making bread today.

A study done experimenting with sourdough fermentation as a means for making wheat bread safe for people with celiac disease had great results.  While the study was small, it did show that individuals with celiac disease who ate specially prepared sourdough wheat bread over the course of 60 days experienced no ill effects.

It was my daughter Maci’s inability to digest wheat that started me on a journey learning about foods that were transformed when they were sprouted or made with sourdough. People who came to my classes and website were experiencing the same results when eating bread that was made with sourdough cultures or sprouted. Even some with Celiac disease seemed to do really well. Now, not everybody who is gluten intolerant can handle it right away. They need to heal their guts first with cultured foods on a regular basis. After this occurs, I have seen so many people thrive when eating breads as long as these breads were fermented or sprouted.

Sourdough bread, fermented for at least 7 hours or longer, is the time it takes to transform the bread. Then it not only easily digested, but often can be handled by those who are gluten intolerant.

http://www.mindbodygreen.com/0-12354/giving-up-gluten-why-you-should-say-hello-to-sourdough.html

Quote
Before the advent of commercial yeast, most bread was made in the “sourdough style,” with its signature cavernous holes. Those holes develop during the process of fermentation, which traditionally happened, at the very least, overnight. During the fermentation process, good bacteria breaks down the gluten proteins, thereby reducing or even eliminating the gluten content all together.

A team of scientists in Italy in 2010 showed that gluten content was much lower in breads that were made in the traditional, old-world style. The difference was so stark that celiacs in the study were able to consume the sourdough with no ill effects. (Could this be why so many of my gluten-sensitive friends do fine on bread and cheese in France or love the pizza in Italy?)

Most commercially made bread in America (even those branded “sourdough”) goes from mix to bread in a matter of hours. Without the longer fermentation time, you may not get the same value. Microbes take time to do their work!


Thus to make it extra sure we've removed all the lectin and gluten from the bread, we should ferment as sour dough for more than 7 hours, overnight, and perhaps up to days. Then to make it extra sure, we can boil the bread for minutes as well as bake it at 500 F!

http://www.motherearthnews.com/real-food/soft-sourdogh-pretzels-recipe-zbcz1402.aspx

Quote
Preheat oven to 425 degrees. Meanwhile, bring a large pan of water to a boil. Carefully add ¼ cup baking soda to the boiling water, it will bubble up.

Gently remove pretzels from baking pan and drop into the boiling water. Simmer about 30 seconds, turn and simmer an additional 30 seconds. Remove with a slotted spoon and return to the baking sheet. Repeat with remaining pretzels.

Sprinkle pretzels with kosher or flake salt, or sesame or poppy seeds. Bake for 20 – 30 minutes or until golden brown.

And we can even pre-bake some baking soda to convert it to reduced form so it can be mixed with hot water to make a substitute for lye to dip the boiled pretzels in before we bake them to give that pretzel coating on the outside:

http://www.splendidtable.org/story/heres-a-substitute-for-lye-when-making-pretzels-at-home

http://www.thekitchn.com/expert-bagel-maker-confirms-you-dont-need-lye-to-make-a-good-bagel-maker-tour-203288


Edit: an all vegan diet is not healthy long-term:

http://paleozonenutrition.com/2011/03/26/why-i-dont-recommend-a-low-fat-raw-vegan-diet/

http://paleozonenutrition.com/2011/03/05/30-bananas-a-day-durianrider-an-analysis-of-his-paleo-vegan-diet/

Quote
How come Harley needs to eat almost double the calories required for his needs? This seems an incredible waste of food resources. He also has a very high fibre intake. Looks like he might spend half the day on the toilet. Don Matesz has an excellent article explaining this:

Quote
Briefly, both of the apes closest to humans by genetic constitution (about 98% identical), chimps and gorillas, are hindgut fermenters.  In chimps and gorillas, the hindgut, or colon, comprises about 52 percent of the total gut volume.  It houses microbes that ferment fiber, converting it to fatty acids that supply up to 65% of the animal’s energy requirements.  In contrast, in humans the hindgut comprises only about 17 percent of total gut volume, and has relatively small microbial population.  At most, microbial fermentation in the hindgut can provide about 10% of human energy requirements.

…it appears that caloric absorption from a completely raw food vegan diet may be as low as 46%.  Given the data I showed above, this means that I would have to eat more than 20 pounds of raw vegetation daily to absorb enough calories to maintain my current body mass and activity levels.

Eating would be a full time job.  Given a 16 hour waking period, I would have to eat 1.25 pounds of food every waking hour to meet my energy needs.   I’d probably spend a good chunk of the time I wasn’t eating sitting on the toilet excreting all the undigested carbage.  My life would consist largely of eating and crapping, just like other primates.

I wonder how much time Harley does spend on the toilet?

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September 14, 2015, 09:51:09 AM
 #1225

Excellent article about cost of living globally:

http://www.allanalytics.com/author.asp?doc_id=276415&_mc=sem_otb_edt_allan

http://www.robslink.com/SAS/democd76/numbeo_price_indices_2015.htm



(click link above to see more charts)

Edit: note the data might not reflect variance within a country, e.g. the USA data is apparently for NYC only.

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September 14, 2015, 11:57:25 AM
 #1226

Excellent article about cost of living globally:

http://www.allanalytics.com/author.asp?doc_id=276415&_mc=sem_otb_edt_allan

http://www.robslink.com/SAS/democd76/numbeo_price_indices_2015.htm



(click link above to see more charts)

Edit: note the data might not reflect variance within a country, e.g. the USA data is apparently for NYC only.

That'll sk(r)ew your results a lot. They could have picked rural Louisiana and skewed it is as much the other way.

I do want to move to Chile though. South American women are extraordinarily schnickity  Shocked 

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September 14, 2015, 12:15:14 PM
 #1227


I do want to move to Chile though. South American women are extraordinarily schnickity  Shocked  

Had to Google it and look what I found:

http://realm-of-eruza.deviantart.com/art/Merry-Xmas-Schnickity-Schick-105449026
LOL! Grin

Chaos could be a form of intelligence we cannot yet understand its complexity.
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September 14, 2015, 12:47:56 PM
 #1228

I do want to move to Chile though. South American women are extraordinarily schnickity  Shocked 

Perschnickity

Pronunciation- Per-shnick-i-ty
A term used to describe an incredibly attractive female. Derived from the Greek root of the word Schnick, meaning beauty, with possible reference to the Greek goddess of beauty herself. The term can be used as both an adjective or a noun.
See also: Schnickity, Schnick, Perschnick
That girl over there is such a perschnick!

Dude, how'd last night go?
Man, she was looking pretty perschnickity!

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September 15, 2015, 03:25:45 AM
 #1229

Thanks for the link, Trollercoaster. This is the kind of thing I was after, though I still have to link a few dots on this subject; to get my head around it. There's a guy in the next village to us with quite an impressive folding HAM radio rig, about 20-30 ft. tall. I'll have to get into conversation.

some dd-wrt capable routers can operate in lower frequencies, the one with atheros chip...but it will only be activated by paying the devs..there is openwrt too..

with computers and routers operating in HAM frequencies we can setup..

Ion towers (individual)

Ion mesh (communities)

Ionosphere (global network)

Ionites  (satellites) Cool


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September 15, 2015, 04:15:39 AM
 #1230

Excellent article about cost of living globally:

http://www.allanalytics.com/author.asp?doc_id=276415&_mc=sem_otb_edt_allan

http://www.robslink.com/SAS/democd76/numbeo_price_indices_2015.htm



(click link above to see more charts)

Edit: note the data might not reflect variance within a country, e.g. the USA data is apparently for NYC only.

That'll sk(r)ew your results a lot.

A lot is an understatement. There are places in the US that are close to 1/10 the cost of living in NYC (that's housing; other costs probably not quite as much of a gap, but still a gap).

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September 15, 2015, 04:19:05 AM
 #1231

Arielbit the problem I found with BBHN & HSMM  is that it's a headache to connect wirelessly to these nodes (maybe easier for non n00bs), there's also project byzantium which I found interesting (I haven't tried it out yet), it looks easier to deploy without needing to find too many pieces of odd hardware or geeks capable of getting it all up and running.
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September 15, 2015, 04:23:58 AM
 #1232

some dd-wrt capable routers

I saw something recently about reprogrammable routers being banned so these may become harder to come by in the future. I don't remember the details.
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September 15, 2015, 04:35:43 AM
 #1233

http://www.extremetech.com/computing/213351-new-fcc-rules-could-ban-dd-wrt-and-router-modification
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September 15, 2015, 10:47:17 AM
Last edit: September 15, 2015, 11:03:54 AM by arielbit
 #1234

some dd-wrt capable routers

I saw something recently about reprogrammable routers being banned so these may become harder to come by in the future. I don't remember the details.

 


Fuck them..another law to ignore, lots of frequencies to hide..

I think it is the frequencies is what they are trying to regulate, it is like banning linux for pc. Manufacturers like buffalo tech releases routers with ddwrt as firmware, asus even advertises some of its routers are ddwrt compatible.

HAM network is an alternative or supplement to the internet for rural areas, when the chips are down these Ion towers will rise.

There is another network that we can look at and possibly utilize for transmitting cryptocoins--> the electrical grid.
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September 16, 2015, 03:48:05 AM
 #1235

http://techcrunch.com/2015/09/15/facebook-agrees-to-do-more-to-help-combat-hate-speech-in-germany/
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September 16, 2015, 06:02:05 AM
 #1236


Just silly. Thinly veiled propaganda machine. What's most shocking to me is how forthright they are in admitting that they steer public opinion with 'counter speech'.

To lighten the thread up a bit:

http://www.independent.co.uk/life-style/gadgets-and-tech/news/commuter-sent-picture-of-mans-penis-in-first-cyberflashing-case-10453890.html

Lucky we have the authorities to run to every time we are offended!




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September 16, 2015, 08:37:51 AM
 #1237

haha  Cool
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September 16, 2015, 03:41:49 PM
 #1238


To lighten the thread up a bit:

http://www.independent.co.uk/life-style/gadgets-and-tech/news/commuter-sent-picture-of-mans-penis-in-first-cyberflashing-case-10453890.html

Lucky we have the authorities to run to every time we are offended!

Classic! A number of things came to mind after reading this article: Was she the only AirDrop female user in the vicinity or did he choose her name from others based on the likelihood of "Lorraine" not being elderly - or for that matter there not being many very young Lorraines around now? Did it fill the screen? And I presume he is likely to have gone out already prepared with the photo, ready to pounce...

I'll leave that one hanging, so to speak.
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September 18, 2015, 02:44:36 AM
 #1239

http://www.theguardian.com/world/2015/sep/17/japan-to-pass-security-bills-despite-protests
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September 18, 2015, 03:08:36 AM
 #1240


what does this mean ? =W=
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